Alzheimer’s disease (AD) is a progressive brain disorder that destroys memory and thinking skills, ultimately causing an inability to perform even simple tasks. It also causes a significant slowdown in a person’s brainwave frequency activity to the point they could be medically described as living in a permanent sleep-like state.

In addition, people with this disease experience atrophy of the brain, which simply means that it is decaying or wasting away. (1) Hence, the old adage, use it or lose it applies to not only brain function, but whether you will have use of your mind, cognition, and memories as you age.


The same thing occurs to your muscles which is medically described as the wasting (thinning) or loss of muscle tissue. The main cause of atrophy is lack of physical activity or disuse (physiologic) of muscles occurs when you don’t use them enough.

As it related to atrophy of the brain, this is what I contend is one of the main causes of dementia and Alzheimer’s Disease worldwide. I believe this wasting away is the result of not actively using your brain on a regular basis to process and learn new information.

The resulting effects of this atrophy allow our microbiome and in particular, fungi to commandeer our cells, neurons, and central nervous system via our gastrointestinal tracts to induce a loss of consciousness or a sleep-like waking state.

As if we have biological laws programmed into our genes that supersede all human-made legal systems and structures that determine our fates. One of these ancient legal codes for not using our brains is to have them slowly eaten from within and wiped clean of all memories attained in this life.

What better and more sinister way than to have the very microbes within our body, blood, and organs be the very legislators of all life?

As it is related to dementia and Alzheimer’s, it is the fungi working within their central nervous system and brains that is turning off certain normal autonomous functions to make them sleep while they are awake in a covert biological attempt to control their minds as it steals their memories.

For example, a 2012 study found that fungal pathogens within the gut microbiota, which are normally well tolerated, may disseminate via the circulation to other sites including the brain leading to a systemic fungal infection, resulting in significant pathology and mortality (Brown et al., 2012).

Several studies have shown that the autopsied brains of Alzheimer’s patients reveal that they are infected with often multiple types of fungi (molds).

Fungal infections have also been widely observed in their blood vessels, which may explain why people who have Alzheimer’s also suffer from vascular pathology.

This is interesting given the fact that it appears to be a memory stealer and brain eater. It makes sense then that fungi may be the culprit responsible for this mind-decaying disease.

But this knowledge has been known by scientists for well over one hundred years.

In 1910, Czech psychiatrist, and an expert on dementia and Alzheimer’s disease, Oskar Fischer, proposed that these diseases were caused by foreign bodies in the brain, most likely fungi, which provoked inflammation and amyloid plaques (see Eikelenboom et al., 2006; Goedert, 2009; Mar 2009 conference news).

In his 1907 paper, Alzheimer described the presence of plaques and tangles in one case of presenile dementia, whereas Fischer described neuritic plaques in 12 cases of senile dementia. These were landmark findings in the history of research in dementia because they delineated the clinicopathological entity that is now known as Alzheimer’s disease.

in 2014, compelling evidence for the existence of fungal proteins in brain samples from Alzheimer’s disease patients. The study titled, “Fungal infection in patients with Alzheimer’s disease,” stated “a variety of fungal species in these samples, dependent on the patient and the tissue tested.

DNA sequencing demonstrated that several fungal species could be found in brain samples. Together, these results show that fungal macromolecules can be detected in the brains of Alzheimer’s disease patients. To our knowledge, these findings represent the first evidence that fungal infection is detectable in brain samples from Alzheimer’s disease patients.” (2)

A 2015 study titled, “Different Brain Regions are Infected with Fungi in Alzheimer’s Disease (AD),” showed the possibility that AD is a fungal disease, or that fungal infection is a risk factor for the disease. The researchers provided evidence in the study that tissue from the central nervous system (CNS) of AD patients contained fungal cells and hyphae.

According to the researchers, “Different brain regions including external frontal cortex, cerebellar hemisphere, entorhinal cortex/hippocampus and choroid plexus contain fungal material, which is absent in brain tissue from control individuals. Analysis of brain sections from ten additional AD patients reveals that all are infected with fungi.

Eleven patients (plus three additional CP samples) were described in this study, as well as in four patients previously analyzed, there is clear evidence for fungal cells inside neurons or extracellularly.

Therefore, 100% of the AD patients analyzed thus far by our laboratory presented fungal cells and fungal material in brain sections.

Moreover, fungal macromolecules (polysaccharides, proteins and DNA) have been found in blood serum from AD patients, and fungal proteins and DNA were detected by proteomic analyses and PCR, respectively, from frozen tissue of AD brain.

Collectively, our findings provide compelling evidence for the existence of fungal infection in the CNS from AD patients, but not in control individuals. (3)

A 2003 study titled, “How a Slime Mold Came to the Aid of Alzheimer’s Research,” details how a structure called a “Hirano body” found in a slime mold is present in increased amounts in Alzheimer’s patients.

It is mostly present in the brain’s major site of learning and memory, the hippocampus.

The study reports, “What causes neurodegenerative diseases like the most common early symptom of Alzheimer’s which is difficulty remembering newly learned information because Alzheimer’s changes typically begin in the part of the brain that affects learning, is still largely unknown, but something destroys nerve cells in the brain over a period of time as victims gradually lose their minds.”

Numerous studies suggest that the accumulation of beta-amyloid peptides (betaAP) plays a central role in the pathogenesis of Alzheimer’s disease. It is well established that betaAP has a wide range of toxic effects on neurons and we can connect their production in the human body to fungi.

The amyloid-beta precursor protein is an important example. It is a large membrane protein that normally plays an essential role in neural growth and repair. However, later in life, a corrupted form can destroy nerve cells, leading to the loss of thought and memory.

Scientists at the Stanford University School of Medicine have shown how the disease is strongly correlated with the overproduction and accumulation of amyloid-β peptide, which begins destroying synapses before it clumps into plaques that lead to nerve cell death.

A 2009 study found that the amyloid-β peptide induced depolarization of skeletal muscle plasma membranes can significantly disturb the functioning of skeletal muscles and therefore contribute to motor dysfunction observed in Alzheimer’s disease and other disorders associated with βAP accumulation.

Depolarization causes the rapid change in membrane potential from a negative to a positive state. The process of depolarization begins with a stimulus like fungi.

Brainwave activity is related to oscillatory activity at different frequencies ranging from 2–4 Hz (delta), 4–8 Hz (theta), 8–13 Hz (alpha), 13–30 Hz (beta), and >30 Hz (gamma). These frequencies transmit certain physiological information on the brain’s functional state during wake and sleep cycles.

The EEG findings of patients with Alzheimer’s disease show a slowing of alpha activity and an increase in slow-frequency activity

A few studies have shown a significant increase in delta and theta power in conjunction with a decrease in alpha and beta power over a period of two years from diagnosis of dementia. In another study, significant increases in delta and theta were found with a decrease in beta, alpha, and mean frequency.

The awakened state is medically described as being in the alpha frequency, which is mainly related to a person’s global attentional readiness. Alpha rhythms represent the dominant resting oscillations of an awakened human brain and have been linked to intelligent quotient, memory, and cognition. (5)


1. Electroencephalographic Rhythms in Alzheimer’s Disease

2. PubMed: Fungal infection in patients with Alzheimer’s disease

3. PubMed: Different Brain Regions are Infected with Fungi in Alzheimer’s Disease

4. PubMed: Different Brain Regions are Infected with Fungi in Alzheimer’s Disease


Pin It on Pinterest